Love you JT but nowhere (even US) have we seen kids super-spreading events
The scientific evidence is out there
Love you JT but nowhere (even US) have we seen kids super-spreading events
The scientific evidence is out there
OK you’re just trolling at this point.
Not getting into a bad faith argument right now. You know this shit is different.
Flattened the curve you say?
How are the countries who reopened schools with new cases at an all-time high doing with that? Could you list them, please?
OK USA, shit will really go upwards when you open schools (it won’t)
Someone open a thread and we’ll have a count of countries and see how it’s going (the countries didn’t open up without better advice than you are getting)
My math says our kids have an insanely higher chance of carrying the disease to their teachers than kids n France do. Therefore, are kids are quite different.
Dear good people of Unstuck:
Please stop responding to Churchill. Thanks in advance.
Sincerely,
Everyone
Counter point: have you ever walked outside in a category 5 hurricane with an umbrella? Then how do you know?
HOLD OUR BEER! MARY POPPINS AIN’T GOT SHIT ON USA#1!!!
55 days of ‘back to school’ and no teachers dying in France
Yea, ours include 12 and 13 year olds.
PSA: stop arguing with Churchill. He has indefensibly horrible views and you will not make any successful traction debating him.
I don’t think people itt are worried about superspreader events or a large upswing in #s. It’s not about that clown. It’s billy joe going to church Sunday and picking it up and giving it to one teacher, one teacher that matters a lot .
Ok I’ll bite. If we were successful in flattening the curve and kept spread where test, trace, and quarantine could work then USA #1 could go back to school. However that’s not what has happened here. We are on pace to knowingly have 0.5% of the population infected over 30 days so in all likelihood 2-3 %.
Would you like me more if I said I was 14
Here’s what you don’t get. Lol USA and everything, but most people here are taking precautions. It’s the covidiots that make headlines. Kids aren’t a super spreader yet because schools have been closed since March. But that goes out the windows if schools open up.
CHIPS ON THE TABLE
300 (positives, no mention of deaths) from 1.3 million (probably the variance on a US covid test)
Are these 300 responsible for the ~8,000 a day Texas cases we are seeing
Look, I’m flogging a dead horse. I apologise for letting you know MANY other countries are doing what you fear without consequence
I highly doubt there are 1.3 million kids in daycare facilities in Texas.
More bad news from the Spain study:
Seroprevalence was 5·0% (95% CI 4·7–5·4) by the point-of-care test and 4·6% (4·3–5·0) by immunoassay, with a specificity–sensitivity range of 3·7% (3·3–4·0; both tests positive) to 6·2% (5·8–6·6; either test positive), with no differences by sex and lower seroprevalence in children younger than 10 years (<3·1% by the point-of-care test). There was substantial geographical variability, with higher prevalence around Madrid (>10%) and lower in coastal areas (<3%). Seroprevalence among 195 participants with positive PCR more than 14 days before the study visit ranged from 87·6% (81·1–92·1; both tests positive) to 91·8% (86·3–95·3; either test positive). In 7273 individuals with anosmia or at least three symptoms, seroprevalence ranged from 15·3% (13·8–16·8) to 19·3% (17·7–21·0). Around a third of seropositive participants were asymptomatic, ranging from 21·9% (19·1–24·9) to 35·8% (33·1–38·5). Only 19·5% (16·3–23·2) of symptomatic participants who were seropositive by both the point-of-care test and immunoassay reported a previous PCR test.
Wasn’t there some hope that a large number of people (1) don’t develop antibodies, (2) have fought the virus off, and (3) therefore might be immune but not showing up in antibody tests?
This doesn’t mean herd immunity is “unachievable.” But it does indicate that we aren’t anywhere near achieving it, and this is evidence against any theory that we’re closer to achieving it than we think.
If I’m understanding the new study correctly, I don’t think it’s inconsistent with that hope. People who tested positive prior to inclusion in this study aren’t a random sample–they would be predominantly people who were symptomatic and disproportionately seriously ill. You really need to do a random sample of people and test for active infection, then follow up later to see if they developed antibodies. That’s assuming that the virus is at detectable levels in the hypothetic naturally-resistant population, of course.